Tiếp theo là một nhà nước hypothyroid với sự trở lại ban đầu sau khoảng 1 năm, mặc dù một số phụ nữ có thể phát triển suy giáp vĩnh viễn. Thường, bướu cổ nontender nhỏ, và các triệu chứng thường nhẹ | 672 MCKEOWN et al develops between 6 weeks to 6 months postpartum with an incidence of about 5 to 10 of all women 12 . This is followed by a hypothyroid state with a return to baseline after about 1 year although some women may develop permanent hypothyroidism. There is usually a small nontender goiter and symptoms are usually mild the result of either the hyperthyroid or hypothyroid state. There is approximately a 70 chance of recurrence in subsequent pregnancies in those who had postpartum thyroiditis with their first pregnancy 12 . Sporadic thyroiditis is a condition similar in clinical course to postpartum thyroiditis but it is not associated with pregnancy 11 . Up to 20 of these patients eventually develop lasting hypothyroidism. Suppurative thyroiditis is an infection of the thyroid gland typically caused by a bacterial infection and less commonly by fungal mycobacterial or parasitic infections 11 . Although rare it is more common in those with pre-existing thyroid disease and in immunocompromised patients. Clinical features include a painful thyroid gland anterior neck pain with erythema fever dysphagia and dysphonia. Amiodarone-induced thyroiditis is associated with either symptomatic or asymptomatic hyperthyroidism. It occurs in about 10 of patients in iodine-deficient areas and less commonly in iodine-replete areas 7 . The mechanism of thyroid dysfunction from the use of amiodarone is caused either by the release of the iodine from the drug itself or by the development of destructive thyroiditis. Individuals with underlying autoimmunity are at increased risk for developing hyperthyroidism if they are taking amiodarone. Nodular and multinodular goiters which are more common in the elderly are a predisposing factor for amiodarone-induced thyroiditis 7 . Clinical features Thyroid hormone affects nearly every organ system in the body. Patients with elevated thyroid hormone present in a hypermetabolic state with signs of increased P-adrenergic activity. .
