báo cáo khoa học: "RIZ1 is potential CML tumor suppressor that is down-regulated during disease progression"

Tuyển tập báo cáo các nghiên cứu khoa học quốc tế ngành y học dành cho các bạn tham khảo đề tài:RIZ1 is potential CML tumor suppressor that is down-regulated during disease progression | Journal of Hematology Oncology BioMed Central Open Access Short report RIZ1 is potential CML tumor suppressor that is down-regulated during disease progression Ashakumary Lakshmikuttyamma1 2 3 Naoto Takahashi1 2 3 Elodie Pastural1 2 3 Emina Torlakovic1 2 3 Hesham M Amin4 5 Guillermo Garcia-Manero4 5 Michael Voralia6 7 Magdalena Czader8 John F DeCoteau1 2 3 and C Ronald Geyer 1 2 3 Address 1Cancer Stem Cell Research Group University of Saskatchewan Saskatoon SK Canada 2Department of Pathology University of Saskatchewan Saskatoon SK Canada 3Department of Biochemistry University of Saskatchewan Saskatoon SK Canada 4Department of Hematopathology MD Anderson Cancer Center University of Texas Houston TX USA 5Department of Leukemia MD Anderson Cancer Center University of Texas Houston TX USA department of Oncology and Hematology Saskatchewan Cancer Agency Saskatoon SK Canada 7Stem Cell Transplant Program Saskatchewan Cancer Agency Saskatoon SK Canada and department of Pathology and Laboratory Medicine Indianapolis IN USA Email Ashakumary Lakshmikuttyamma - ashakum@ Naoto Takahashi - naotot@ Elodie Pastural - EminaTorlakovic - emt323@ Hesham M Amin - hamin@ Guillermo Garcia-Manero - ggarciam@ Michael Voralia - Magdalena Czader - mczader@ John F DeCoteau - C Ronald Geyer - Corresponding author Published 14 July 2009 Received 17 March 2009 Journal of Hematology Oncology 2009 2 28 doi 1756-8722-2-28 Accepted 14 July 2009 This article is available from http content 2 1 28 2009 Lakshmikuttyamma et al licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License http licenses by which permits unrestricted use distribution and reproduction in any medium provided the original work is .

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