Tuyển tập báo cáo các nghiên cứu khoa học quốc tế ngành y học dành cho các bạn tham khảo đề tài: Chest pain with ST segment elevation in a patient with prosthetic aortic valve infective endocarditis: a case report | JOURNALOFMEDICAL CASE reports Chest pain with ST segment elevation in a patient with prosthetic aortic valve infective endocarditis a case report Luther et al. Luther et al. Journal of Medical Case Reports 2011 5 408 http content 5 1 408 24 August 2011 BioMed Central Luther et al. Journal of Medical Case Reports 2011 5 408 http content 5 1 408 JOURNAL OF MEDICAL CASE REPORTS CASE REPORT Open Access Chest pain with ST segment elevation in a patient with prosthetic aortic valve infective endocarditis a case report Vishal Luther1 Refai Showkathali2 and Reto Gamma2 Abstract Introduction Acute ST-segment elevation myocardial infarction secondary to atherosclerotic plaque rupture is a common medical emergency. This condition is effectively managed with percutaneous coronary intervention or thrombolysis. We report a rare case of acute myocardial infarction secondary to coronary embolisation of valvular vegetation in a patient with infective endocarditis and we highlight how the management of this phenomenon may not be the same. Case presentation A 73-year-old British Caucasian man with previous tissue aortic valve replacement was diagnosed with and treated for infective endocarditis of his native mitral valve. His condition deteriorated in hospital and repeat echocardiography revealed migration of vegetation to his aortic valve. Whilst waiting for surgery our patient developed severe central crushing chest pain with associated anterior ST segment elevation on his electrocardiogram. Our patient had no history or risk factors for ischaemic heart disease. It was likely that coronary embolisation of part of the vegetation had occurred. Thrombolysis or percutaneous coronary intervention treatments were not performed in this setting and a plan was made for urgent surgical intervention. However our patient deteriorated rapidly and unfortunately died. Conclusion Clinicians need to be aware that atherosclerotic plaque rupture
