Tuyển tập báo cáo các nghiên cứu khoa học quốc tế ngành y học dành cho các bạn tham khảo đề tài:Early bare-metal stent thrombosis presenting with cardiogenic shock: a case report | Lampropoulos et al. Journal of Medical Case Reports 2011 5 509 http content 5 1 509 JOURNAL OF MEDICAL CASE REPORTS CASE REPORT Open Access Early bare-metal stent thrombosis presenting with cardiogenic shock a case report Konstantinos M Lampropoulos1 2 Themistoklis A Iliopoulos 1 and Werner Budts2 Abstract Introduction Although stents have improved the safety and efficacy of percutaneous coronary interventions coronary stent thrombosis remains a serious complication. Case presentation We present the case of a 64-year-old Caucasian man from Greece with symptoms and electrocardiographic findings suggestive of acute inferior myocardial infarction who complained of chest pain and rapidly developed cardiogenic shock 48 hours after primary percutaneous coronary intervention. Conclusion The most common cause of early bare-metal stent thrombosis is stent malapposition. Intravascular ultrasound is the preferred method to recognize predictors of coronary events that are not detected by angiography. Introduction Stents have improved the safety and efficacy of percutaneous coronary interventions PCI by reducing acute or imminent vessel closure and by reducing restenosis rates compared with conventional balloon angioplasty 1 . In addition coronary vasomotor reactivity has been found intact after stent implantation and long-term clinical and angiographic follow-up have attested to the durability of their action 2 . Nevertheless coronary stent thrombosis remains a serious complication of PCI. Case presentation A 64-year-old male Caucasian patient was admitted to our hospital with clinical and electrocardiographical findings suggesting acute inferior myocardial infarction. Our patient had a history of hypertension and dyslipidemia but was not taking any medication at the time of admission. Laboratory findings were suggestive of acute cardiac ischemia. His plasma levels of N-terminal pro-B-type natriuretic troponin I creatine kinase and creatine kinase .
