báo cáo khoa học: "Cerebral amyloidoma mimicking intracranial tumor: a case report"

Tuyển tập báo cáo các nghiên cứu khoa học quốc tế ngành y học dành cho các bạn tham khảo đề tài: Cerebral amyloidoma mimicking intracranial tumor: a case report | Landau et al. Journal of Medical Case Reports 2010 4 308 http content 4 1 308 jAc JOURNALOF medical ÌỤr case REPORTS CASE REPORT Open Access Cerebral amyloidoma mimicking intracranial tumor a case report Danny Landau Nicholas Avgeropoulos Joe Ma Abstract Introduction Cerebral amyloidoma is an infrequently recognized condition that can be confused with a more malignant etiology. Few cases have been reported. We present a case report and a review of the literature. Case presentation Our patient was a 64-year-old Caucasian man who was incidentally discovered to have a brain mass. He was found to have a cerebral amyloidoma. Conclusion After discovery of the true etiology of his brain abnormality it was determined that our patient had a more benign disease than was initially feared. Cases such as this demonstrate why consideration of this disorder is important. Introduction Cerebral amyloidomas are rare entities infrequently described in the medical literature. Most commonly they are noticed incidentally on brain scans. Frequently they are confused with primary brain neoplasms. The clinical course tends to be benign although long-term data is lacking. Case report Our patient was a 64-year-old Caucasian man with a medical history of chronic obstructive pulmonary disease coronary artery disease and hyperlipidemia. He had been traveling and experienced a transient loss of consciousness lasting a few seconds while in an airport abroad. He sought medical attention and an initial computed tomography CT study of his head without contrast revealed a lobular hyperdensity in the right posterior periventricular distribution tracking to the adjacent parietal subcortical white matter. A follow-up magnetic resonance imaging MRI study revealed an infiltrating lesion extending from the right peritrigonal area across the corona radiata and into the subcortical white matter. The lesion showed increased T2-weighted and low T1-weighted signal intensity with .

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