Tuyển tập báo cáo các nghiên cứu khoa học quốc tế ngành y học dành cho các bạn tham khảo đề tài: Abnormal coactivation of knee and ankle extensors is related to changes in heteronymous spinal pathways after stroke | I JOURNAL OF NEUROENGINEERING AND REHABILITATION Abnormal coactivation of knee and ankle extensors is related to changes in heteronymous spinal pathways after stroke Dyer et al. Dyer et al. Journal of NeuroEngineering and Rehabilitation 2011 8 41 http content 8 1 41 2 August 2011 2 BioMed Central Dyer et al. Journal of NeuroEngineering and Rehabilitation 2011 8 41 http content 8 1 41 Iril JOURNAL OF NEUROENGINEERING NCR AND REHABILITATION RESEARCH Open Access Abnormal coactivation of knee and ankle extensors is related to changes in heteronymous spinal pathways after stroke Joseph-Omer Dyer Eric Maupas Sibele de Andrade Melo Daniel Bourbonnais and Robert Forget Abstract Background Abnormal coactivation of leg extensors is often observed on the paretic side of stroke patients while they attempt to move. The mechanisms underlying this coactivation are not well understood. This study 1 compares the coactivation of leg extensors during static contractions in stroke and healthy individuals and 2 assesses whether this coactivation is related to changes in intersegmental pathways between quadriceps and soleus Sol muscles after stroke. Methods Thirteen stroke patients and ten healthy individuals participated in the study. Levels of coactivation of knee extensors and ankle extensors were measured in sitting position during two tasks maximal isometric voluntary contractions in knee extension and in plantarflexion. The early facilitation and later inhibition of soleus voluntary EMG evoked by femoral nerve stimulation were assessed in the paretic leg of stroke participants and in one leg of healthy participants. Results Coactivation levels of ankle extensors mean SEM 56 7 of Sol EMG max and of knee extensors 52 10 of vastus lateralis VL EMG max during the knee extension and the ankle extension tasks respectively were significantly higher in the paretic leg of stroke participants than in healthy participants 26 5
