báo cáo khoa học: " Two alternative recessive quantitative trait loci influence resistance to spring black stem and leaf spot in Medicago truncatula"

Tuyển tập báo cáo các nghiên cứu khoa học quốc tế ngành y học dành cho các bạn tham khảo đề tài: Two alternative recessive quantitative trait loci influence resistance to spring black stem and leaf spot in Medicago truncatula | BMC Plant Biology BioMed Central Research article Two alternative recessive quantitative trait loci influence resistance to spring black stem and leaf spot in Medicago truncatula Lars G Kamphuis1 Judith Lichtenzveig1 2 Richard P Oliver1 and Simon R Ellwood 1 Address Australian Centre for Necrotrophic Fungal Pathogens State Agricultural Biotechnology Centre Murdoch University Perth 6150 Western Australia Australia and 2Commonwealth Scientific and Industrial Research Organisation Plant Industry Private Bag No. 5 Wembley 6913 Western Australia Australia Email Lars G Kamphuis - Judith Lichtenzveig - Richard P Oliver - Simon R Ellwood - Corresponding author Open Access Published 26 March 2008 Received II December 2007 BMC Plant Biology 2008 8 30 doi 1471-2229-8-30 Accepted 26 March 2008 This article is available from http 1471-2229 8 30 2008 Kamphuis et al licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License http licenses by which permits unrestricted use distribution and reproduction in any medium provided the original work is properly cited. Abstract Background Knowledge of the genetic basis of plant resistance to necrotrophic pathogens is incomplete and has been characterised in relatively few pathosystems. In this study the cytology and genetics of resistance to spring black stem and leaf spot caused by Phoma medicaginis an economically important necrotrophic pathogen of Medicago spp. was examined in the model legume M. truncatula. Results Macroscopically the resistant response of accession SA27063 was characterised by small hypersensitive-like spots following inoculation while the susceptible interaction with accessions A17 and SA3054 showed necrotic lesions and spreading chlorosis. No unique cytological differences were observed .

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