Tuyển tập báo cáo các nghiên cứu khoa học quốc tế ngành hóa học dành cho các bạn yêu hóa học tham khảo đề tài: Temporal expression and cellular origin of CC chemokine receptors CCR1, CCR2 and CCR5 in the central nervous system: insight into mechanisms of MOG-induced EAE | Journal of Neuroinflammation BioMed Central Research Temporal expression and cellular origin of CC chemokine receptors CCR1 CCR2 and CCR5 in the central nervous system insight into mechanisms of MOG-induced EAE Sana Eltayeb1 Anna-Lena Berg 2 Hans Lassmann3 Erik Wallstrom1 Maria Nilsson4 Tomas Olsson1 Anders Ericsson-Dahlstrand4 and Dan Sunnemark4 Open Access Address Department of Clinical Neuroscience Center for Molecular Medicine Neuroimmunology Unit Karolinska Institute S-171 76 Stockholm Sweden 2Department of Pathology Safety Assessment AstraZeneca R D Sodertalje S-15185 Sodertalje Sweden 3Brain Research Institute University of Vienna Vienna Austria and 4Department of Disease Biology Local Discovery Research Area CNS and Pain Control AstraZeneca R D Sodertalje S-151 85 Sodertalje Sweden Email Sana Eltayeb - Anna-Lena Berg - Hans Lassmann - Erik Wallstrom - Maria Nilsson - Tomas Olsson - Anders Ericsson-Dahlstrand - Dan Sunnemark - Corresponding author Published 7 May 2007 Received 5 February 2007 Journal of Neuroinflammation 2007 4 14 doi 1742-2094-4-14 Accepted 7 May 2007 This article is available from http content 4 1 14 2007 Eltayeb et al licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License http licenses by which permits unrestricted use distribution and reproduction in any medium provided the original work is properly cited. Abstract Background The CC chemokine receptors CCR1 CCR2 and CCR5 are critical for the recruitment of mononuclear phagocytes to the central nervous system CNS in multiple sclerosis MS and other neuroinflammatory diseases. Mononuclear phagocytes are effector cells capable of .
