Nicotinic acetylcholine receptors (nAChRs) have been reported to be overexpressed in malignancies in humans and is associated with tumorigenesis and cell migration. In previous studies of gastric cancer, alpha7 nicotinic acetylcholine receptor (α7-nAChR) overexpression leads to epithelial-mesenchymal transition (EMT) and promotes the migration of gastric cancer cells. | Migration of gastric cancer is suppressed by recombinant Newcastle disease virus rL-RVG via regulating α7-nicotinic acetylcholine receptors ERK- EMT
