Tuyển tập các báo cáo nghiên cứu về y học được đăng trên tạp chí y học Critical Care giúp cho các bạn có thêm kiến thức về ngành y học đề tài: Cerebral haemodynamics and carbon dioxide reactivity during sepsis syndrome. | Available online http content 11 6 R123 Research Cerebral haemodynamics and carbon dioxide reactivity during sepsis syndrome Christof Thees1 Markus Kaiser1 Martin Scholz1 Alexander Semmler2 Michael T Heneka3 Georg Baumgarten1 Andreas Hoeft4 and Christian Putensen5 Department of Anaesthesiology and Intensive Care Medicine University of Bonn 53105 Bonn Germany 2Department of Neurology University of Bonn 53105 Bonn Germany 3Department of Neurology University of Bonn 53105 Bonn Germany 4Department of Anaesthesiology and Intensive Care Medicine University of Bonn 53105 Bonn Germany 5Department of Anaesthesiology and Intensive Care Medicine University of Bonn 53105 Bonn Germany Corresponding author Christof Thees Received 8 May 2007 Revisions requested 12 Jun 2007 Revisions received 20 Oct 2007 Accepted 28 Nov 2007 Published 28 Nov 2007 Critical Care 2007 11 R123 doi cc6185 This article is online at http content 11 6 R1 23 2007 Thees et al licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License http licenses by 20 which permits unrestricted use distribution and reproduction in any medium provided the original work is properly cited. Open Access Abstract Background Most patients with sepsis develop potentially irreversible cerebral dysfunctions. It is yet not clear whether cerebral haemodynamics are altered in these sepsis patients at all and to what extent. We hypothesized that cerebral haemodynamics and carbon dioxide reactivity would be impaired in patients with sepsis syndrome and pathological electroencephalogram patterns. Methods After approval of the institutional ethics committee 10 mechanically ventilated patients with sepsis syndrome and pathological electroencephalogram patterns underwent measurements of cerebral blood flow and jugular venous oxygen saturation before and after reduction of the arterial .
