Tuyển tập các báo cáo nghiên cứu về y học được đăng trên tạp chí y học Critical Care giúp cho các bạn có thêm kiến thức về ngành y học đề tài: CIDE-A is expressed in liver of old mice and in type 2 diabetic mouse liver exhibiting steatosis. | Comparative Hepatology BioMed Central Research CIDE-A is expressed in liver of old mice and in type 2 diabetic mouse liver exhibiting steatosis Bruce Kelder 1 Keith Boyce2 4 Andres Kriete2 5 Ryan Clark1 Darlene E Berryman6 Sheila Nagatomi2 Edward O List1 Mark Braughler2 7 and John J Kopchick1 3 Open Access Address 1Edison Biotechnology Institute Ohio University Athens OH 45701 USA 2Clinical Data Inc Newton MA 02458 USA 3Department of Biomedical Sciences College of Osteopathic Medicine Ohio University Athens OH 45701 USA 4Immune Tolerance Network Pittsburgh PA 15238 USA 5Drexel University and Coriell Bioinformatics Initiative School of Biomedical Engineering Drexel University Philadelphia PA 19104 USA 6School of Human and Consumer Sciences Ohio University Athens OH 45701 USA and 7Rheogene Norristown PA 19403 USA Email Bruce Kelder - kelder@ Keith Boyce - kboyce@ Andres Kriete - akriete@ Ryan Clark - ryanclark80@ Darlene E Berryman - berrymad@ Sheila Nagatomi - sheilanagatomi@ Edward O List - edlist@ Mark Braughler - mbraughler@ John J Kopchick - kopchick@ Corresponding author Published I May 2007 Received 19 July 2006 Comparative Hepatology 2007 6 4 doi 1476-5926-6-4 Accepted 1 May 2007 This article is available from http content 6 1 4 2007 Kelder et al licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License http licenses by which permits unrestricted use distribution and reproduction in any medium provided the original work is properly cited. Abstract Background Increased levels of circulating fatty acids caused by insulin resistance and increased adipocyte lipolysis can accumulate within the liver resulting in steatosis. This steatosis sensitizes the liver to inflammation and further injury which can lead to liver dysfunction. We .
