Tuyển tập các báo cáo nghiên cứu về y học được đăng trên tạp chí y học Critical Care giúp cho các bạn có thêm kiến thức về ngành y học đề tài: Mechanical ventilation during experimental sepsis increases deposition of advanced glycation end products and myocardial inflammation. | Available online http content 13 3 R87 Research Mechanical ventilation during experimental sepsis increases deposition of advanced glycation end products and myocardial inflammation Martin CJ Kneyber1 2 3 Roel P Gazendam1 2 Hans WM Niessen2 4 5 Jan-Willem Kuiper1 6 2 Claudia C Dos Santos6 Arthur S Slutsky6 and Frans B Plotz1 2 Department of Pediatric Intensive Care VU university medical center . Box 7057 1007 MB Amsterdam The Netherlands institute for Cardiovascular Research ICaR-VU VU university medical center . Box 7057 1007 MB Amsterdam The Netherlands 3Beatrix Children s Hospital University Medical Center . Box 30001 9700 RB Groningen The Netherlands department of Pathology VU university medical center . Box 7057 1007 MB Amsterdam The Netherlands department of Cardiac Surgery VU university medical center . Box 7057 1007 MB Amsterdam The Netherlands 6Keenan Research Centre Li Ka Shing Knowledge Institute St. Michael s Hospital 30 Bond Street Toronto Ontario M5B 1W8 University of Toronto Toronto Ontario Canada Corresponding author Martin CJ Kneyber Received 2 Apr 2009 Revisions requested 24 Apr 2009 Revisions received 2 Jun 2009 Accepted 9 Jun 2009 Published 9 Jun 2009 Critical Care 2009 13 R87 doi cc7911 This article is online at http content 13 3 R87 2009 Kneyber et al. licensee BioMed Central Ltd. This is an open access article distributed under the terms of the Creative Commons Attribution License http licenses by which permits unrestricted use distribution and reproduction in any medium provided the original work is properly cited. Open Access Abstract Introduction Increasing evidence links advanced glycation end products AGE including Ne- carboxymethyl lysine CML to the development of heart failure. Accumulation of AGE leads to myocardial inflammation which is considered as one of the possible mechanisms underlying sepsis-induced cardiac dysfunction. We .
