Báo cáo y học: " Abdominal Compartment Syndrome: pathophysiology and definitions"

Tuyển tập các báo cáo nghiên cứu về y học được đăng trên tạp chí y học quốc tế cung cấp cho các bạn kiến thức về ngành y đề tài: Abdominal Compartment Syndrome: pathophysiology and definitions | Scandinavian Journal of Trauma Resuscitation and Emergency Medicine BioMed Central Review Abdominal Compartment Syndrome pathophysiology and definitions Michael L Cheatham Open Access Address Department of Surgical Education Orlando Regional Medical Center Orlando Florida 32806 USA Email Michael L Cheatham - Published 2 March 2009 Received 8 February 2009 Scandinavian Journal of Trauma Resuscitation and Emergency Medicine 2009 17 10 doi 1757-7241-17-10 Accepted 2 March 2009 This article is available from http content 17 1 10 2009 Cheatham licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License http licenses by which permits unrestricted use distribution and reproduction in any medium provided the original work is properly cited. Abstract Intra-abdominal hypertension the presence of elevated intra-abdominal pressure and abdominal compartment syndrome the development of pressure-induced organ-dysfunction and failure have been increasingly recognized over the past decade as causes of significant morbidity and mortality among critically ill surgical and medical patients. Elevated intra-abdominal pressure can cause significant impairment of cardiac pulmonary renal gastrointestinal hepatic and central nervous system function. The significant prognostic value of elevated intra-abdominal pressure has prompted many intensive care units to adopt measurement of this physiologic parameter as a routine vital sign in patients at risk. A thorough understanding of the pathophysiologic implications of elevated intra-abdominal pressure is fundamental to 1 recognizing the presence of intraabdominal hypertension and abdominal compartment syndrome 2 effectively resuscitating patients afflicted by these potentially life-threatening diseases and 3 preventing the development of intra-abdominal pressure-induced end-organ dysfunction .

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