Tuyển tập các báo cáo nghiên cứu về y học được đăng trên tạp chí y học Wertheim cung cấp cho các bạn kiến thức về ngành y đề tài: Prevalence of reverse transcriptase and protease mutations associated with antiretroviral drug resistance among drug-naïve HIV-1 infected pregnant women in Kagera and Kilimanjaro regions, Tanzania. | AIDS Research and Therapy BioMed Central Open Access Prevalence of reverse transcriptase and protease mutations associated with antiretroviral drug resistance among drug-náive HIV-I infected pregnant women in Kagera and Kilimanjaro regions Tanzania Balthazar M Nyombi 1 2 3 Carol Holm-Hansen2 4 Knut I Kristiansen3 Gunnar Bjune2 and Fredrik Muller3 Address Research Laboratory Kilimanjaro Christian Medical College Moshi Tanzania international Community Health Institute of General Practice and Community Medicine Faculty of Medicine University of Oslo Oslo Norway Institute of Microbiology Faculty of Medicine University of Oslo and Rikshospitalet University Hospital Oslo Norway and 4Division of Infectious Disease Control Norwegian Institute of Public Health Oslo Norway Email Balthazar M Nyombi - bnyombi@ Carol Holm-Hansen - Knut I Kristiansen - Gunnar Bjune - Fredrik Muller - Corresponding author Published 21 June 2008 Received 20 February 2008 AIDS Research and Therapy 2008 5 13 doi l742-6405-5-l3 Accepted 21 June 2008 This article is available from http content 5 l l3 2008 Nyombi et al licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License http licenses by which permits unrestricted use distribution and reproduction in any medium provided the original work is properly cited. Abstract Background Access to antiretroviral drugs for HIV-l infection has increased in sub-Saharan Africa SSA during the past few years. Mutations in the HIV-l genome are often associated with treatment failure as indicated by viral replication and elevated levels of virus in the blood. Mutations conferring resistance to antiretroviral drugs are based on comparing gene sequences with corresponding consensus sequences of HIV-l subtype B that .
