Tuyển tập các báo cáo nghiên cứu về y học được đăng trên tạp chí y học Critical Care cung cấp cho các bạn kiến thức về ngành y đề tài: The effect of interruption to propofol sedation on auditory event-related potentials and electroencephalogram in intensive care patients. | Available online http content 8 6 R483 Research The effect of interruption to propofol sedation on auditory event-related potentials and electroencephalogram in intensive care patients Heidi Ypparila1 Silvia Nunes2 Ilkka Korhonen3 Juhani Partanen4 and Esko Ruokonen5 Open Access Department of Clinical Neurophysiology Kuopio University Hospital and Department of Applied Physics University of Kuopio Kuopio Finland department of Anesthesiology and Intensive Care Division of Intensive Care Kuopio University Hospital Kuopio Finland 3Professor VTT Information Technology Tampere Finland 4Professor Department of Clinical Neurophysiology Kuopio University Hospital Kuopio Finland department of Anesthesiology and Intensive Care Division of Intensive Care Kuopio University Hospital Kuopio Finland Corresponding author Heidi Yppărilă Received 19 May 2004 Revisions requested 23 August 2004 Revisions received 7 September 2004 Accepted 23 September 2004 Published 22 October 2004 Critical Care 2004 8 R483-R490 DOI cc2984 This article is online at http content 8 6 R483 2004 Yppărilă et al. licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License http licenses by which permits unrestricted use distribution and reproduction in any medium provided the original work is cited. Abstract Introduction In this observational pilot study we evaluated the electroencephalogram EEG and auditory event-related potentials ERPs before and after discontinuation of propofol sedation in neurologically intact intensive care patients. Methods Nineteen intensive care unit patients received a propofol infusion in accordance with a sedation protocol. The EEG signal and the ERPs were measured at the frontal region Fz and central region Cz both during propofol sedation and after cessation of infusion when the sedative effects had subsided. The EEG signal was .
