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Báo cáo y học: "Parthenolide inhibits ERK and AP-1 which are dysregulated and contribute to excessive IL-8 expression and secretion in Cystic Fibrosis cells"

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Tuyển tập báo cáo các nghiên cứu khoa học quốc tế ngành y học dành cho các bạn tham khảo đề tài: Parthenolide inhibits ERK and AP-1 which are dysregulated and contribute to excessive IL-8 expression and secretion in Cystic Fibrosis cells. | Journal of Inflammation BioMed Central Vz The Open Access Publisher This Provisional PDF corresponds to the article as it appeared upon acceptance. Fully formatted PDF and full text HTML versions will be made available soon. Parthenolide inhibits ERK and AP-1 which are dysregulated and contribute to excessive IL-8 expression and secretion in Cystic Fibrosis cells Journal of Inflammation 2011 8 26 doi 10.1186 1476-9255-8-26 Aicha Saadane aicha.saadane@case.edu Jean Eastman jean.eastman@case.edu Melvin Berger melvin.berger@uhhostitals.com Tracey L Bonfield tracey.bonfield@case.edu ISSN 1476-9255 Article type Research Submission date 13 October 2010 Acceptance date 12 October 2011 Publication date 12 October 2011 Article URL http www.journal-inflammation.com content 8 1 26 This peer-reviewed article was published immediately upon acceptance. It can be downloaded printed and distributed freely for any purposes see copyright notice below . Articles in Journal of Inflammation are listed in PubMed and archived at PubMed Central. For information about publishing your research in Journal of Inflammation or any BioMed Central journal go to http www.journal-inflammation.com authors instructions For information about other BioMed Central publications go to http www.biomedcentral.com 2011 Saadane etal. licensee BioMed Central Ltd. This is an open access article distributed under the terms of the Creative Commons Attribution License http creativecommons.org licenses by 2.0 which permits unrestricted use distribution and reproduction in any medium provided the original work is properly cited. Parthenolide inhibits ERK and AP-1 which are dysregulated and contribute to excessive IL-8 expression and secretion in cystic fibrosis cells Aicha Saadanet Jean Eastman Melvin Berger and Tracey L. Bonfield Department of Pediatrics Case Western Reserve University 11100 Euclid Avenue BRB-822 Cleveland Ohio 44106 OH 44106 Phone 216 368-4376. Fax 216 368-4223 tCorresponding author .

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