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Báo cáo y học: " Epigallocatechin-3-gallate (EGCG) inhibits the migratory behavior of tumor bronchial epithelial cells"

Tuyển tập các báo cáo nghiên cứu về y học được đăng trên tạp chí y học 'Respiratory Research cung cấp cho các bạn kiến thức về ngành y đề tài: " Epigallocatechin-3-gallate (EGCG) inhibits the migratory behavior of tumor bronchial epithelial cells. | Respiratory Research BioMed Central Research Epigallocatechin-3-gallate EGCG inhibits the migratory behavior of tumor bronchial epithelial cells Salma Hazgui1 Arnaud Bonnomet1 Beatrice Nawrocki-Raby1 Magali Milliot1 Christine Terryn2 Jérôme Cutrona1 2 Myriam Polette1 3 Philippe Birembaut1 3 and Jean-Marie Zahm 1 Open Access Address 1INSERM UMRS903 Reims F-51092 France 2Univ Reims Champagne Ardenne IFR53 Reims F-51100 France and 3CHU Reims Hôpital Maison Blanche Laboratoire Pol Bouin Reims F-51092 France Email Salma Hazgui - salma.hazgui@univ-reims.fr Arnaud Bonnomet - arnaud.bonnomet@univ-reims.fr Beatrice Nawrocki- Raby - beatrice.raby@univ-reims.fr Magali Milliot - magali.milliot@univ-reims.fr Christine Terryn - christine.terryn@univ-reims.fr Jérôme Cutrona - jerome.cutrona@univ-reims.fr Myriam Polette - myriam.polette@univ-reims.fr Philippe Birembaut - pbirembaut@chu-reims.fr Jean-Marie Zahm - jm.zahm@univ-reims.fr Corresponding author Published 21 April 2008 Received 24 July 2007 Respiratory Research 2008 9 33 doi 10.1186 1465-9921-9-33 Accepted 21 April 2008 This article is available from http respiratory-research.cOm content 9 1 33 2008 Hazgui et al licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License http creativecommons.org licenses by 2.0 which permits unrestricted use distribution and reproduction in any medium provided the original work is properly cited. Abstract Background Many studies associated the main polyphenolic constituent of green tea - -Epigallocatechin-3-gallate EGCG with inhibition of cancers invasion and metastasis. To date most of the studies have focused on the effect of EGCG on cell proliferation or death. Since cell migration is an important mechanism involved in tumor invasion the aim of the present work was to target another approach of the therapeutic effect of EGCG by .