Lung fibrotic tenascin-C upregulation is associated with other extracellular matrix proteins and induced by TGFβ1

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Lung fibrotic tenascin-C upregulation is associated with other extracellular matrix proteins and induced by TGFβ1

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Idiopathic pulmonary fibrosis (IPF) is a progressive parenchymal lung disease of unknown aetiology and poor prognosis, characterized by altered tissue repair and fibrosis. The extracellular matrix (ECM) is a critical component in regulating cellular homeostasis and appropriate wound healing. |

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Lung fibrotic tenascin-C upregulation is associated with other extracellular matrix proteins and induced by TGFβ1

Idiopathic pulmonary fibrosis (IPF) is a progressive parenchymal lung disease of unknown aetiology and poor prognosis, characterized by altered tissue repair and fibrosis. The extracellular matrix (ECM) is a critical component in regulating cellular homeostasis and appropriate wound healing. |

“Velcro-type” crackles predict specific radiologic features of fibrotic interstitial lung disease

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The anti-fibrotic agent pirfenidone synergizes with cisplatin in killing tumor cells and cancer-associated fibroblasts

Pirfenidone as salvage treatment for refractory bleomycin-induced lung injury: A case report of seminoma

Diaphragmatic ultrasound findings correlate with dyspnea, exercise tolerance, health-related quality of life and lung function in patients with fibrotic interstitial lung disease

Plakoglobin expression in fibroblasts and its role in idiopathic pulmonary fibrosis

Symptom prevalence of patients with fibrotic interstitial lung disease: A systematic literature review

A multicentre retrospective observational study on Polish experience of pirfenidone therapy in patients with idiopathic pulmonary fibrosis: The PolExPIR study

Báo cáo y học: "Altered expression of membrane-bound and soluble CD95/Fas contributes to the resistance of fibrotic lung fibroblasts to FasL induced apoptosis"

The elevation of serum napsin A in idiopathic pulmonary fibrosis, compared with KL-6, surfactant protein-A and surfactant protein-D

Đang chuẩn bị liên kết để tải về tài liệu: Lung fibrotic tenascin-C upregulation is associated with other extracellular matrix proteins and induced by TGFβ1

Idiopathic pulmonary fibrosis (IPF) is a progressive parenchymal lung disease of unknown aetiology and poor prognosis, characterized by altered tissue repair and fibrosis. The extracellular matrix (ECM) is a critical component in regulating cellular homeostasis and appropriate wound healing. |

“Velcro-type” crackles predict specific radiologic features of fibrotic interstitial lung disease

The impact of high-flow nasal cannula oxygen therapy on exercise capacity in fibrotic interstitial lung disease: A proof-ofconcept randomized controlled crossover trial

The anti-fibrotic agent pirfenidone synergizes with cisplatin in killing tumor cells and cancer-associated fibroblasts

Pirfenidone as salvage treatment for refractory bleomycin-induced lung injury: A case report of seminoma

Diaphragmatic ultrasound findings correlate with dyspnea, exercise tolerance, health-related quality of life and lung function in patients with fibrotic interstitial lung disease

Plakoglobin expression in fibroblasts and its role in idiopathic pulmonary fibrosis

Symptom prevalence of patients with fibrotic interstitial lung disease: A systematic literature review

A multicentre retrospective observational study on Polish experience of pirfenidone therapy in patients with idiopathic pulmonary fibrosis: The PolExPIR study

Báo cáo y học: "Altered expression of membrane-bound and soluble CD95/Fas contributes to the resistance of fibrotic lung fibroblasts to FasL induced apoptosis"

The elevation of serum napsin A in idiopathic pulmonary fibrosis, compared with KL-6, surfactant protein-A and surfactant protein-D

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Lung fibrotic tenascin-C upregulation is associated with other extracellular matrix proteins and induced by TGFβ1