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Báo cáo y học: "Therapies, School of Biological and Biomedical Sciences, Griffith University, Brisbane 4111, Australia. †The Institute for Molecular Biosciences"

Tuyển tập các báo cáo nghiên cứu về y học được đăng trên tạp chí y học Minireview cung cấp cho các bạn kiến thức về ngành y đề tài: Therapies, School of Biological and Biomedical Sciences, Griffith University, Brisbane 4111, Australia. †The Institute for Molecular Biosciences. | Open Access Research Alternate transcription of the Toll-like receptor signaling cascade Christine A WellsH Alistair M ChalkH Alistair Forrest Darrin Taylor Nic Waddelh Kate Schroder S Roy Himes Geoffrey Faulkner Sandra Lo Takeya Kasukawa Hideya Kawaji Chikatoshi Kai Jun Kawai Shintaro Katayama Piero Carninci Yoshihide Hayashizaki David A Humet and Sean M Grimmond Addresses Eskitis Institute for Cell and Molecular Therapies School of Biological and Biomedical Sciences Griffith University Brisbane 4111 Australia. The Institute for Molecular Biosciences The University of Queensland Brisbane 4072 Australia. Karolinska Institutet S-171 77 Stockholm Sweden. Genome Exploration Research Group Genome Network Project Core Group RIKEN Genomic Sciences Center RIKEN Yokohama Institute Yokohama Kanagawa 230-0045 Japan. Genome Science Laboratory Discovery Research Institute RIKEN Wako Institute Wako Saitama 351-0198 Japan. The Special Research Centre for Functional and Applied Genomics The University of Queensland St Lucia 4072 Australia. n These authors contributed equally to this work. Correspondence Christine A Wells. Email c.wells@griffith.edu.au Published 17 February 2006 Genome Biology 2006 7 R10 doi 10.1 186 gb-2006-7-2-r10 The electronic version of this article is the complete one and can be found online at http genomebiology.com 2006 7 2 R10 Received 4 October 2005 Revised 9 December 2005 Accepted 16 January 2006 2006 Wells et al. licensee BioMed Central Ltd. This is an open access article distributed under the terms of the Creative Commons Attribution License http creativecommons.org licenses by 2.0 which permits unrestricted use distribution and reproduction in any medium provided the original work is properly cited. Abstract Background Alternate splicing of key signaling molecules in the Toll-like receptor Tlr cascade has been shown to dramatically alter the signaling capacity of inflammatory cells but it is not known how common this mechanism is. We provide .