Stat3 is a positive regulator of gap junctional intercellular communication in cultured, human lung carcinoma cells

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Stat3 is a positive regulator of gap junctional intercellular communication in cultured, human lung carcinoma cells

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Neoplastic transformation of cultured cells by a number of oncogenes such as src suppresses gap junctional, intercellular communication (GJIC); however, the role of Src and its effector Signal transducer and activator of transcription-3 (Stat3) upon GJIC in non small cell lung cancer (NSCLC) has not been defined. |

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Stat3 is a positive regulator of gap junctional intercellular communication in cultured, human lung carcinoma cells

báo cáo khoa học: "Prognostic significance of STAT3 and phosphorylated STAT3 in human soft tissue tumors - a clinicopathological analysis"

Unphosphorylated STAT3 in heterochromatin formation and tumor suppression in lung cancer

Prognostic value of protein inhibitor of activated STAT3 in breast cancer patients receiving hormone therapy

Berberine suppresses tumorigenicity and growth of nasopharyngeal carcinoma cells by inhibiting STAT3 activation induced by tumor associated fibroblasts

STAT3 polymorphism and Helicobacter pylori CagA strains with higher number of EPIYA-C segments independently increase the risk of gastric cancer

báo cáo khoa học: "Adenovirus-mediated delivery of bFGF small interfering RNA reduces STAT3 phosphorylation and induces the depolarization of mitochondria and apoptosis in glioma cells U251"

Báo cáo y học: "High nuclear expression of STAT3 is associated with unfavorable prognosis in diffuse large B-cell lymphoma"

báo cáo khoa học: " High-throughput sequencing identifies STAT3 as the DNA-associated factor for p53-NF-κB-complex-dependent gene expression in human heart failure"

báo cáo hóa học: " Inhibitory effect of a tyrosine-fructose Maillard reaction product, 2,4-bis(p-hydroxyphenyl)-2butenal on amyloid-b generation and inflammatory reactions via inhibition of NF-B and STAT3 activation in cultured astrocytes and microglial BV-2 cells"

Báo cáo khoa học: The reactive oxygen species–Src–Stat3 pathway provokes negative feedback inhibition of apoptosis induced by high-fluence low-power laser irradiation

Đang chuẩn bị liên kết để tải về tài liệu: Stat3 is a positive regulator of gap junctional intercellular communication in cultured, human lung carcinoma cells

báo cáo khoa học: "Prognostic significance of STAT3 and phosphorylated STAT3 in human soft tissue tumors - a clinicopathological analysis"

Unphosphorylated STAT3 in heterochromatin formation and tumor suppression in lung cancer

Prognostic value of protein inhibitor of activated STAT3 in breast cancer patients receiving hormone therapy

Berberine suppresses tumorigenicity and growth of nasopharyngeal carcinoma cells by inhibiting STAT3 activation induced by tumor associated fibroblasts

STAT3 polymorphism and Helicobacter pylori CagA strains with higher number of EPIYA-C segments independently increase the risk of gastric cancer

báo cáo khoa học: "Adenovirus-mediated delivery of bFGF small interfering RNA reduces STAT3 phosphorylation and induces the depolarization of mitochondria and apoptosis in glioma cells U251"

Báo cáo y học: "High nuclear expression of STAT3 is associated with unfavorable prognosis in diffuse large B-cell lymphoma"

báo cáo khoa học: " High-throughput sequencing identifies STAT3 as the DNA-associated factor for p53-NF-κB-complex-dependent gene expression in human heart failure"

báo cáo hóa học: " Inhibitory effect of a tyrosine-fructose Maillard reaction product, 2,4-bis(p-hydroxyphenyl)-2butenal on amyloid-b generation and inflammatory reactions via inhibition of NF-B and STAT3 activation in cultured astrocytes and microglial BV-2 cells"

Báo cáo khoa học: The reactive oxygen species–Src–Stat3 pathway provokes negative feedback inhibition of apoptosis induced by high-fluence low-power laser irradiation

Đang chuẩn bị liên kết để tải về tài liệu:
Stat3 is a positive regulator of gap junctional intercellular communication in cultured, human lung carcinoma cells

báo cáo hóa học: " Inhibitory effect of a tyrosine-fructose Maillard reaction product, 2,4-bis(p-hydroxyphenyl)-2butenal on amyloid-b generation and inflammatory reactions via inhibition of NF-B and STAT3 activation in cultured astrocytes and microglial BV-2 cells"