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báo cáo hóa học: " Lipopolysaccharide induced inflammation in the perivascular space in lungs"

Tuyển tập báo cáo các nghiên cứu khoa học quốc tế ngành hóa học dành cho các bạn yêu hóa học tham khảo đề tài:Lipopolysaccharide induced inflammation in the perivascular space in lungs | Journal of Occupational Medicine and Toxicology BioMed Central Open Access Lipopolysaccharide induced inflammation in the perivascular space in lungs Thomas Tschernig 11 Kyathanahalli S Janardhant2 3 Reinhard Pabst1 and Baljit Singh2 Address 1Dept. of Functional and Applied Anatomy -4120- Medical School of Hannover Carl-Neuberg-Str. 1 30625 Hannover Germany 2Immunology Research Group Departments of Veterinary Biomedical Sciences and Veterinary Microbiology Western College of Veterinary Medicine University of Saskatchewan 52 Campus Drive Saskatoon SK S7N 5B4 Canada and 3Diagnostic Medicine and Pathobiology 1800 Denison Avenue Kansas State University Manhattan Kansas 66506 USA Email Thomas Tschernig - tschernig.thomas@mh-hannover.de Kyathanahalli S Janardhan - kjanard@vet.k-state.edu Reinhard Pabst - pabst.reinhard@mh-hannover.de Baljit Singh - baljit.singh@usask.ca Corresponding author tEqual contributors Published 30 July 2008 Received 29 March 2007 Journal of Occupational Medicine and Toxicology 2008 3 17 doi 10.1186 1745-6673-3-17 Accepted 30 July 2008 This article is available from http www.occup-med.cOm content 3 1 17 2008 Tschernig et al licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License http creativecommons.org licenses by 2.0 which permits unrestricted use distribution and reproduction in any medium provided the original work is properly cited. Abstract__ Background Lipopolysaccharide LPS contained in tobacco smoke and a variety of environmental and occupational dusts is a toxic agent causing lung inflammation characterized by migration of neutrophils and monocytes into alveoli. Although migration of inflammatory cells into alveoli of LPS-treated rats is well characterized the dynamics of their accumulation in the perivascular space PVS leading to a perivascular inflammation PVI of pulmonary arteries .