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Báo cáo hóa học: "Neutrophil depletion reduces edema formation and tissue loss following traumatic brain injury in mice"

Tuyển tập báo cáo các nghiên cứu khoa học quốc tế ngành hóa học dành cho các bạn yêu hóa học tham khảo đề tài: Neutrophil depletion reduces edema formation and tissue loss following traumatic brain injury in mice | Journal of Neuroinflammation BioMed Central Vz The Open Access Publisher This Provisional PDF corresponds to the article as it appeared upon acceptance. Fully formatted PDF and full text HTML versions will be made available soon. Neutrophil depletion reduces edema formation and tissue loss following traumatic brain injury in mice Journal of Neuroinflammation 2012 9 17 doi 10.1186 1742-2094-9-17 Ellinor Kenne ellinor.kenne@ki.se Anna Erlandsson anna.erlandsson@neuro.uu.se Lennart Lindbom lennart.lindbom@ki.se Lars Hillered lars.hillered@neuro.uu.se Fredrik Clausen fredrik.clausen@neuro.uu.se ISSN 1742-2094 Article type Research Submission date 23 September 2011 Acceptance date 23 January 2012 Publication date 23 January 2012 Article URL http www.ineuroinflammation.com content 9 1 17 This peer-reviewed article was published immediately upon acceptance. It can be downloaded printed and distributed freely for any purposes see copyright notice below . Articles in JNI are listed in PubMed and archived at PubMed Central. For information about publishing your research in JNI or any BioMed Central journal go to http www.jneuroinflammation.com authors instructions For information about other BioMed Central publications go to http www.biomedcentral.com 2012 Kenne etal. licensee BioMed Central Ltd. This is an open access article distributed under the terms of the Creative Commons Attribution License http creativecommons.org licenses by 2.0 which permits unrestricted use distribution and reproduction in any medium provided the original work is properly cited. Neutrophil depletion reduces edema formation and tissue loss following traumatic brain injury in mice Ellinor Kenne1 Anna Erlandsson2 Lennart Lindbom1 Lars Hillered2 Fredrik Clausen2 1 Department of Physiology and Pharmacology Karolinska Institutet Stockholm Sweden 2 Department of Neuroscience Neurosurgery Uppsala University Uppsala University Hospital Uppsala Sweden. Email addresses for authors EK ellinor.kenne@ki.se .